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Clinical and Diagnostic Laboratory Immunology, November 2001, p. 1263-1266, Vol. 8, No. 6
1071-412X/01/$04.00+0   DOI: 10.1128/CDLI.8.6.1263-1266.2001
Copyright © 2001, American Society for Microbiology. All rights reserved.

Chlamydia pneumoniae Exacerbates Aortic Inflammatory Foci Caused by Murine Cytomegalovirus Infection in Normocholesterolemic Mice

Katalin Burian,1 Klara Berencsi,2 Valeria Endresz,1 Zsofia Gyulai,1 Tibor Valyi-Nagy,3 Istvan Valyi-Nagy,4 Marta Bakay,1 Yuemei Geng,2 Dezso Virok,1 Laszlo Kari,2 Rozalia Hajnal-Papp,5 Giorgio Trinchieri,2 and Eva Gonczol1,2,*

Department of Medical Microbiology1 and Department of Pathology,5 University of Szeged, Szeged, and First Department of Internal Medicine, Semmelweis University, Budapest,4 Hungary; The Wistar Institute, Philadelphia, Pennsylvania2; and Department of Pathology, Vanderbilt University, Nashville, Tennessee3

Received 26 February 2001/Returned for modification 9 May 2001/Accepted 18 July 2001

Inflammatory foci induced by murine cytomegalovirus infection in normocholesterolemic mice were present temporarily in the aortic wall, but some of these foci developed into advanced lesions that persisted late after infection. The early foci induced by virus infection were significantly exacerbated following a single inoculation with Chlamydia pneumoniae.

* Corresponding author. Mailing address: The Wistar Institute, 3601 Spruce St., Philadelphia, PA 19104. Phone: (215) 898-3887. Fax: (215) 898-3868. E-mail: Gonczol{at}wistar.upenn.edu.

Clinical and Diagnostic Laboratory Immunology, November 2001, p. 1263-1266, Vol. 8, No. 6
1071-412X/01/$04.00+0   DOI: 10.1128/CDLI.8.6.1263-1266.2001
Copyright © 2001, American Society for Microbiology. All rights reserved.




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Antimicrob. Agents Chemother. Clin. Microbiol. Rev. Infect. Immun.
J. Clin. Microbiol. J. Virol. ALL ASM JOURNALS

Copyright © 2001 by the American Society for Microbiology. All rights reserved.